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Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas

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PDF) Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas

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Pathophysiological involvement of host mitochondria in SARS-CoV-2 infection that causes COVID-19: a comprehensive evidential insight

Regulation of type I interferon responses by mitochondria-derived reactive oxygen species in plasmacytoid dendritic cells - ScienceDirect

Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling

Mitochondrial reactive oxygen species regulate the induction of CD8+ T cells by plasmacytoid dendritic cells

Mitochondrial Functions in Infection and Immunity: Trends in Cell Biology

Mitochondria-mediated oxidative stress during viral infection: Trends in Microbiology