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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Last updated 29 março 2025
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDF) Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial dysfunction in COVID-19: an overview of evidence
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Frontiers Molecular mechanism of VE-cadherin in regulating
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial barrier dysfunction in septic shock - Opal - 2015
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
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