2′-O methylation of the viral mRNA cap evades host restriction by IFIT family members
Por um escritor misterioso
Last updated 04 abril 2025


Attenuation of SARS‐CoV‐2 replication and associated inflammation by concomitant targeting of viral and host cap 2'‐O‐ribose methyltransferases

2'-O-Methylation of the second transcribed nucleotide within the mRNA 5' cap impacts the protein production level in a cell-specific manner and contributes to RNA immune evasion. - Abstract - Europe PMC

Viruses, Free Full-Text
2′-O Methylation of the Viral mRNA Cap by West Nile Virus Evades Ifit1-Dependent and -Independent Mechanisms of Host Restriction In Vivo

Ribose 2′-O-methylation provides a molecular signature for the distinction of self and non-self mRNA dependent on the RNA sensor Mda5

IFITs, once upregulated due to IFN signaling, play various roles in

Targeting cap1 RNA methyltransferases as an antiviral strategy - ScienceDirect

The mRNA Cap 2′-O-Methyltransferase CMTR1 Regulates the Expression of Certain Interferon-Stimulated Genes

IFIT1: A dual sensor and effector molecule that detects non-2'-O methylated viral RNA and inhibits its translation. - Abstract - Europe PMC

PDF) 2'-O-Methylation of the viral mRNA cap evades host restriction by IFIT family members

IJMS, Free Full-Text

Messenger RNA Cap Methylation in Vesicular Stomatitis Virus, a Prototype of Non‐Segmented Negative‐Sense RNA Virus

Frontiers The role of post-transcriptional regulation in SARS-CoV-2 infection and pathogenicity
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