RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL

Regulated Necrosis - The Linkermann Lab

Shikonin overcomes drug resistance and induces necroptosis by regulating the miR-92a-1-5p/MLKL axis in chronic myeloid leukemia

Canonical versus noncanonical necrosome activation by three distinct

RIPK3 Activates Parallel Pathways of MLKL-Driven Necroptosis and FADD-Mediated Apoptosis to Protect against Influenza A Virus - ScienceDirect

From (Tool)Bench to Bedside: The Potential of Necroptosis Inhibitors

RIP3 is an upregulator of aerobic metabolism and the enhanced respiration by necrosomal RIP3 feeds back on necrosome to promote necroptosis

RIPK1 and RIPK3 Kinases Promote Cell-Death-Independent Inflammation by Toll-like Receptor 4 - ScienceDirect

MLKL in cancer: more than a necroptosis regulator. - Abstract - Europe PMC

Transient knockdown of components along the RIPK1–RIPK3–MLKL–PGAM5–Drp1

MLKL in cancer: more than a necroptosis regulator